Dodging a bullet
For around 2 months, my wife and I lowered our bed and slept at 3.8 degrees, around half the normal incline for us, after reading from others that they found it was suiting them better than five degrees or more. So sounded like a useful experiment. In hindsight, I wish we hadn't, because I was shivering and we both developed severe leg cramps, My calf muscles felt like they were twisting and my foot was turning in. We experienced very poor sleep, frequent bathroom trips at night, cold hands and feet that didn't warm quickly, generally colder in bed too. Though initially we both slept sounder, but woke up feeling very un-rested. It was as if we hadn't had any sleep.
We felt lethargic during the day, but in the interest of science continued with the lower incline until I began to struggle on walks. Breathing felt like the air was acidic and burning my lungs, the air was very cold and damp. I experienced crushing pain in chest with left arm aching, jaw aching and had to sit five times to rest on way back to car.
When I got home, I took a teaspoon full of cayenne pepper in a glass of fruit juice. I'm pretty sure this stopped the heart attack from progressing further because the relief was almost instantaneous. (I had read about this a few years back) The bed and the sofa are now back to usual incline.
This experience told me I was probably having a heart attack.
Some interesting history
Many years before, we had lost a dog through heart attack on beach in exactly the same oppressive air and weather conditions. He was only 5 and was running around, keeled over and was gone within seconds. So I knew through my work on circulation and my experience working in Buckfastleigh, a river valley where the air was heavy and instantly drained all of our energy leaving us soaked in sweat and extremely lethargic. Which completely vanished immediately we left the valley and we were all back to normal. The locals have given it a name, calling it Buckfastis and on such days there is an exodus from the valley with people unable to endure the heavy laboured breathing.
This link with high humidity fits perfectly with IBT theory. Our lungs need to evaporate water as we exhale in order to alter our blood density and drive our circulation. Breathing in saturated air will adversely affect it!
Prior to having respiratory distress and severe chest pain, on the 4th I had a blood test at Dr's, which caused a painful visible swelling near injection site. The nurse didn't take enough blood so had to go back for another blood test on the 6th, where I told the nurse I had severe chest pains and pain down my left arm, particularly at the injection site, which was swollen and I had aching in jaw. The nurse said that's because of your blood pressure. I was sent away, but knew I was in trouble and didn't trust the nurses judgement. So I tried to call our GP but constantly engaged. It was now the weekend so I didn't manage to get through until 9th December. Told my Dr the same as I had told the nurse and was asked if I could get to the hospital A&E as soon as possible.
By now, the aching and pain had subsided a lot and the inclined bed was helping a great deal by calming everything down, but still there was a problem with my breathing, night sweats and aching, albeit somewhat milder and eased by our now correctly inclined bed.
We arrived in A&E and I must say, that despite being very busy, the attention I received was very good. I had a cannula inserted and various tests were done. I was then moved to a Cardiac / chest pain ward for 5 days.
It was fascinating watching people from all walks of life who were experiencing the same problems that I was. The good news is, finally got my blood pressure under control with “special drugs” that according to the cardiologist don't damage kidneys.
I'm pretty sure that lowering the bed as mentioned above, was probably a cause.
Anyway, I remained inclined at 5 degrees for my stay in hospital, and managed to bend the ears of doctors and nurses, and a few patients, as you can imagine.
One Doctor agreed with my theory and thought it fascinating. Most of the nurses could see the logic in it.
I had no pain whatsoever and recovered very fast on IBT in hospital. Though I did experience some aching in chest and arm while having hot showers. There’s that high humidity connection again!
I was allowed to walk around the ward and visit bathroom on my own.
The cardiologist came to talk to me about my options and told me he would need to introduce a colourless odourless liquid into my arteries and that it would negatively affect my renal function.
I replied, would that be gadolinium? He replied yes. I said; No thanks, it will trash my already damaged kidneys and I will end up on dialysis for life. He agreed it would damage my renal function and could completely shut it down.
There where two types of surgery on offer. One was an angioplasty, where a catheter is inserted into an arm artery or groin artery and fed to heart vessels, It can inflate to open up arteries and has a camera giving constant visual feedback. A dye containing gadolinium is then introduced via the catheter, which shows the flow through various vessels to identify a site for inserting a stent.
The problem here for me and many others is that it is highly toxic and destructive to the kidneys, brain and other organs which would have completely shut my kidneys down and led to dialysis for life. So no thanks.
A triple bypass, where veins are cut from legs and used to replace clogged arteries, again the dye would be used, so no thanks. https://en.wikipedia.org/wiki/Gadolinium Furthermore, the transplanted veins which then behave like arteries, are subjected to the same problem with platelets causing narrowing, so it’s a buy some time operation rather than a remedial intervention.
My decision appears to have been a reasonable call: https://www.verywellhealth.com/do-angioplasty-and-stents-prolong-life-4021221
One of the patients I met was happy to show me his scars on his legs, where is veins were removed and chest where his rib cage was prized apart. He also shared his concerns that he was still having angina pains after his surgery. Yet tried his utmost to convince me to go down the same route?
According to a nurse in the ward, over 90% of the population have some issues with clogged arteries, many are a ticking time bomb. One young lady in there had lost two sisters in their 20's to angina attacks and she was in there getting sorted after she experienced the same symptoms.
I asked the surgeon what had they used to do before catheters and the gadolinium dye? He said we gave statins, blood thinners and advise on fish oil supplements--which apparently do clear out fatty deposits. No more cheese (Doh) as this is the wrong type of fats, though not giving up butter, but will moderate it.
Smokers are the main source of inmates in the cardio ward. So Jude has had lectures from me about it.
I was asked which drugs I had been on, and reeled off a long list. He replied: “I would never have given a patient with kidney problems any of those drugs.” And assured me that the drugs he was giving me were completely safe and in his words, did not enter the kidneys, which I thought was a strange thing to say at the time? I told him that I would be researching all of the drugs when I went home.
At least someone had now confirmed my suspicions that drugs I had been prescribed for high blood pressure had caused further decline in my renal function. I have no doubt that most of the damage done to my kidneys was because of the drugs prescribed by my GP’s and renal consultant.
Apparently my heart is now operating at 38% ejection fraction.
Normal ejection fraction is (EF= 55-60%). Article on this: https://myheart.net/articles/heart-function-including-ejection-fraction-ef/
The drugs prescribed by the cardiologist were implicated in causing renal failure and premature deaths. He had lied to my face and was obviously very good at it!
Betablockers for example have caused over 800,000 deaths in 5 years in Europe alone. https://www.telegraph.co.uk/news/worldnews/1952818/Betablockers-have-caused-800000-deaths.html
My experience was that my fingers and toes would go blue, then turn white, with oxygen sats down to 72 about 30+ minutes after taking bisoprolol and this would subside over several hours.
Aspirin damages kidneys! https://www.ncbi.nlm.nih.gov/pubmed/10643705/
Statins damage kidneys!
I discussed with the cardiologist that the heart attack came while out walking in very cold damp air. Felt like I was breathing in acid, which was burning my lungs, followed by crushing pain in my chest, pain down left arm was excruciating and pain in jaw, with swollen glands in throat. I told the cardiologist and asked if it was common to have an attack in cold damp weather? He said yes it certainly is and is very common.
1. For several months I had reduced the amount of water that I was drinking. \i was drinking a huge amount on the advice of Doctors, consultants and nurses, who advised me to drink plenty of water consistently. How much is too much and what damage was I doing by drinking several litres a day on top of beverages?
2. I had been drinking Kefir, which is a fermented full fat milk. Prior to heart attack, I was consuming Kefir, which is a bacterial and yeast culture made from whole milk. I guess I was probably causing the inflammation by overloading with bacteria, good or bad it get's into the blood by releasing it's waste products through the intestines. Add to this the lactic acid in kefir and it's a possibilty in understanding why I got into trouble. Especially when I'm lactose intolerant. I thought that the bacteria and yeast would have rendered the milk harmless to me, guess I got that wrong.
Ironically, I wrote to Lucyane on the 5th of Dec telling her I had been sleeping at 3.8 degrees to see what would happen and decided to revert back to 6.5 degrees as was experiencing leg cramps, poor sleep, feeling cold in bed and going bathroom too much
Beta blocker Experience: Letter to Dr
Dear Dr xxxxxxxxxxxxx Sent: 27 December 2019 13:27
I have been getting some odd circulation issues that occur after taking my morning medications, which are getting progressively worse each day, with hands changing colour as if all blood has been drained from fingers. Numbness and tingling in toes and fingers. Looks like Raynauds Syndrome. Also getting odd heart beats intermittently. All of these symptoms ease off later in the day and none return after evening meds. So logically, it is a drug that I take in the morning, so after quick research, it does appear to by bisoprolol fumarate that is causing the problems and I will not be taking this medication again. I do remember having problems with a betablocker in the past and had to stop taking the drug.
It also appears to negatively affect renal function, which the hospital cardiologist assured me it wouldn't. "
The acute effects of bisoprolol 10 mg i.v., a new beta1-selective adrenoceptor antagonist, on heart rate, mean blood pressure (mBP), glomerular filtration rate (GFR), para-aminohippuric acid clearance (CPAH), sodium clearance, urine volume and plasma renin activity (PRA), were studied in 6 patients with essential hypertension. Heart rate decreased by 23%, mBP remained unchanged, and GFR decreased by 14% and CPAH by 23%. PRA was depressed on average by 25%. Urine volume and sodium clearance also declined by 9 and 13%, respectively, but the changes were not statistically significant. The fall in heart rate was significantly correlated with that in GFR and CPAH. Changes in GFR were correlated significantly with those in CPAH. The acute changes in renal function induced by bisoprolol are considered to be due to a reduction in cardiac output and increased systemic vascular resistance."
Giannetta, E., Feola, T., Gianfrilli, D. et al. Is chronic inhibition of phosphodiesterase type 5 cardioprotective and safe? A meta-analysis of randomized controlled trials. BMC Med 12, 185 (2014) doi:10.1186/s12916-014-0185-3
“R E S EARCH A R TIC L E Is chronic inhibition of phosphodiesterase type 5 cardioprotective and safe? A meta-analysis of randomized controlled trials Elisa Giannetta1 , Tiziana Feola1 , Daniele Gianfrilli1 , Riccardo Pofi1 , Valentina Dall’Armi2 , Roberto Badagliacca3 , Federica Barbagallo1 , Andrea Lenzi1 and Andrea M Isidori1* Abstract Background: The myocardial effects of phosphodiesterase type 5 inhibitors (PDE5i) have recently received consideration in several preclinical studies. The risk/benefit ratio in humans remains unclear. Methods: We performed a meta-analysis of randomized, placebo-controlled trials (RCTs) to evaluate the efficacy and safety of PDE5i on cardiac morphology and function. From March 2012 to December 2013 (update: May 2014), we searched English-language studies from MEDLINE, EMBASE, Cochrane Central Register of Controlled Trials and SCOPUS-selecting RCTs of continuous PDE5i administration that reported cardiovascular outcomes: cardiac geometry and performance, afterload, endothelial function and safety. The pooled estimate of a weighted mean difference between treatment and placebo was obtained for all outcomes using a random effects model. A test for heterogeneity was performed and the I2 statistic calculated. Results: Overall, 1,622 subjects were treated, with 954 randomized to PDE5i and 772 to placebo in 24 RCTs. According to our analysis, sustained PDE5 inhibition produced: (1) an anti-remodeling effect by reducing cardiac mass (−12.21 g/m2 , 95% confidence interval (CI): −18.85; −5.57) in subjects with left ventricular hypertrophy (LVH) and by increasing end-diastolic volume (5.00 mL/m2 ; 95% CI: 3.29; 6.71) in non-LVH patients; (2) an improvement in cardiac performance by increasing cardiac index (0.30 L/min/m2 , 95% CI: 0.202; 0.406) and ejection fraction (3.56%, 95% CI: 1.79; 5.33). These effects are parallel to a decline of N-terminal-pro brain natriuretic peptide (NT-proBNP) in subjects with severe LVH (−486.7 pg/ml, 95% CI: −712; -261). PDE5i administration also produced: (3) no changes in afterload parameters and (4) an improvement in flow-mediated vasodilation (3.31%, 95% CI: 0.53; 6.08). Flushing, headache, epistaxis and gastric symptoms were the commonest side effects. Conclusions: This meta-analysis suggests for the first time that PDE5i have anti-remodeling properties and improve cardiac inotropism, independently of afterload changes, with a good safety profile. Given the reproducibility of the findings and tolerability across different populations, PDE5i could be reasonably offered to men with cardiac hypertrophy and early stage heart failure. Given the limited gender data, a larger trial on the sex-specific response to long-term PDE5i treatment is required. Keywords: Phosphodiesterase-5, Heart failure, Hypertension, Endothelial function, Cardiac remodeling, Cardiac hypertrophy, Erectile dysfunction, Sildenafil, Pulmonary hypertension “
Case history brief: Viagra for Angina (Author unknown)
Sep 01, 2018 I was diagnose with clogged arteries over a year ago. I even had a stent put in because one of my arteries was ecluded. I still had stable angina every time I did some for of cardio activity or is I ate something with sodium. I started using Nitroglycerin patches and even have the pills just in case my angina episode occurs.
I had a date and I read online that you can't take viagra and any nitrates cause it can drop your blood pressure really low. So I stopped using my nitroglycerin for about 48 hours so I can take the Viagra. I noticed that I wasn't having chest pains a few hours after I took the viagra. Then I didn't have angina for over a day. Once I felt a little chest pain, I took another viagra and no angina. I've been taken viagra for over 2 weeks and no angina. I even have been doing cardio with no chest pain whatsoever. I told my cardiologist this, but she says that it was not good and I should stop immediately and start using the nitrates again. But she didn't give me a good reason to stop. I have no angina whatsoever. I was wondering has anyone else tried this? If so, have you found any pros or cons? I can't find anything and I've read that viagra was originally to prevent angina. I want to keep taking the viagra but I don't want to damage the heart either, Help please. Thanks
Add a comment
Who Is Andrew K Fletcher?
For more than 23 years, Andrew, 60 years young, has worked tirelessly, selflessly, and self-funded. He has been helping people around the world regain control of their health by simply advising them to raise their beds at the head end to a five-degree angle.
This is based on his new understanding of the role that gravity plays in driving our circulation.
As a free-thinking child, Andrew would take things apart, needing to understand how it all works. He did the same with the science text books, which he states will never look the same again. He openly admits to being a difficult, stubborn student questioning lessons and tutors, rather than accepting subject content as facts. Andrew left school early to enter a career as a vehicle mechanic, later moving into heavy engineering as a fabricator welder, and obtained many other skills from a wide range of disciplines.
Andrew’s love of the natural world and problem solving was fuelled through absorbing countless Open University Programmes (OUP) about nature, history, science, medicine, art and engineering. As a moralist, philosopher and a humanist Andrew has much to say about poverty and inequality throughout the world.
How Inclined Bed Therapy (IBT) Started
Odd as it may seem, Andrew’s insatiable appetite for problem solving was aroused by a television programme addressing irrigation problems in deserts in 1994. The Operation OASIS (Overseas Arid Soil Irrigation Solution) project found that soil was made too salty by high evaporation, effectively rendering soils unproductive. However, in the backdrop from the camera footage, Andrew noticed that the trees appeared to be unaffected by the same irrigated water.
He asked: What are trees doing with salts and minerals?
To answer this problem Andrew obtained a textbook: McKean, DG. GCSE Biology. Hodder Murray, 1989. McKean was very critical of the explanations of sap flow and lack of supporting evidence for fluid transportation in plants and trees. These unconvincing descriptions prompted Andrew to re-examine the facts and ignore all unsubstantiated assumptions of a one-way flow to the tree’s canopy.
Andrew reasoned that if a tree stood vertically it was using gravity in its long tubular structures. If it was not using gravity it would grow horizontally! It is a fact that 98% of the water entering through the tree’s roots evaporates through the leaves. This process is called transpiration.
Andrew applied his knowledge and experience of siphons and non—pumped hot water systems to build up a hypothesis. The simple flow and return mechanism of a non-pumped hot water system relies upon density changes. Heat from a boiler causes water to rise. The transfer of heat from a tubular coil inside a tank to the hot water supply causes the water inside the coil to cool, making it denser, generating an efficient gravity driven return flow.
Recently, his experiment at Brixham, Devon, where he caused water to flow vertically up 24 meters in a single open ended tube, was verified in Nature Journal, under Scientific Reports, after three doctors tested it to 15 meters to try to determine the height limit of a siphon, which is believed to be 10 metres. Initially, no recognition was given to Andrew. This has since been corrected in a Corrigendum, attached to the original publication.
Sap Circulation Discovery
Andrew then applied the analogy of a simple flow and return system to the tree. The tree is warmed by the sun and transfers heat to the tree’s long continuous tube cell structures. It made sense.
Realising that for sap density changes to take place and initiate circulation, it would require the down flow to be denser than the return flow. McKean confirmed this to be the case in his book but this explanation was not sufficient to explain observed sap flow rates.
Andrew concluded that the leaves producing sugar by converting carbon dioxide in a process called photosynthesis combined with dissolved nutrients and minerals from the soil water, increases sap density in the leaves.
Evaporation from the leaves serves to further increase sap density by the same amount of water loss from transpiration. Transpiration is the process of water movement to the atmosphere, from aerial parts such as leaves, stems and flowers, by evaporation.
Therefore, soil water entering the tree from the roots contains a dissolved, dilute solution of minerals and nutrients, dilutes sap density. Dilute sap flows up the tree transported in the xylem vessels , which are long thin non-living tubes, powered by the loading and concentration of sap with sugars produced by the leaves which flows down in the aptly named phloem vessels to a sink (lower reciprocal).
The sink is always the path of least resistance and can be fruits, where sugars are stored. Or by being taken up in the trees continual cycle of expansion and growth in the trunk and branches, providing the tree with a means to offload sugars and nutrients from the heavier downward flowing sap, ultimately reaching the roots under a positive pressure. (Think of sap flow behaving like a liquid plunger in a syringe, creating a positive pressure in front and a lower pressure behind it)
Having off loaded sugars and nutrients on it's journey down the tree, the sap becomes less dense and is ultimately re-diluted by incoming water from the soil into the roots, pulled into the tree by the lower pressure generated in the xylem which is also under applied tension, caused by the down-flow.
For every action there must be a reaction! A down-flow must cause a return flow! This is how trees and plants circulate sap!
Cohesion is the property of liquid molecules bonding together and reaches out beyond the sap in the roots, connecting with soil water molecules, pulling water into the roots. Therefore, the downward flowing sap inevitably causes the less dense sap to rise because sap molecules are linked together through cohesion . (Think of water in sap behaving as liquid rubber).
Osmosis, root pressure and capillary action are effectively redundant in his theory.
Andrew’s Eureka Moments
Tall trees now made sense! Instead of asking how a one-way flow from roots to leaves operates, which has baffled scientists for hundreds of years, because they asked the wrong question, we now have a robust circulation theory for plants and trees, supported by many experiments with plastic tubes, showing how gravity can raise water to great heights.
The Birth of Inclined Bed Therapy (IBT)
Almost instantly, Andrew, who thinks laterally, made the connection with gravity for our own circulation systems. He hypothesised that there must be an imbalance in the density of our blood and other bodily fluids-due to evaporation from our lungs, upper respiratory tract, sinuses, skin and eyes, all of which excrete water to the atmosphere and therefore must concentrate the fluids from which evaporation originates.
He obtained more medical and science text books to see what was already known and, just as he found a wholly inadequate explanation for sap flow in trees, he found inadequate explanations for the origination of human circulation. Andrew also found that there was little research or understanding into how primary circulation occurs, long before the heart is formed, it beats in a developing embryo. A Pulsate flow is visible in the vessels that develop into the heart. in fact there were contradictions and doubts about all circulations.
Andrew’s Logical Approach Appears to Fit Well with Blood Flow
Andrew’s hypothesis argues that respiration in the lungs must concentrate the blood because of the water lost due to evaporation. So, the blood exiting the lungs is denser than the blood entering the lungs and therefore must contribute to circulation.
Homo-Erectus (When we Stood Vertical)
Being upright now meant so much more to Andrew, because homo sapiens were obviously benefiting a great deal more than anyone had realised. Our vertical posture was indeed a very smart move in our evolutionary path.
By standing on our own two feet, we got ahead of other species due to this boost in our circulation. This argument raises many more interesting questions about how our intelligence surpassed that of primates and many other species. Dean Falk and Professor Michel Cabanac in the radiator theory, http://deanfalk.com/wp-content/uploads/2012/06/1990-Falk-Brain-evolution-in-Homo-The-radiator-theory.pdf raised interesting observations. Falk studied early human skulls and noted that the holes in the back of the skull, which accommodated blood vessels to and from the brain, had migrated to the top of the skull when we stood upright.
Cabanac discovered that blood flow from the brain reverses against the flow from the heart, when our body temperature is raised through exercise. https://link.springer.com/article/10.1007/BF02335925?no-access=true
What this tells us is that our circulation changed permanently in the upright posture.
Andrew deduced that blood density from higher evaporation, due to exercise, changed the direction of flow and must have played a significant role in the relocation of these vessels over generations, indicating a remapping of vessels may well have contributed to our thinking patterns.
Andrew’s question was obvious:
If gravity was more important than anyone could have imagined, why are we ignoring it by sleeping horizontally in bed?
The direction of blood flow through the long tubular vessels in our body, which run from head to toe, made Andrew question whether flat bed-rest was efficient in replenishing and maintaining the human body.
Andrew’s questioning of why we all sleep on flat beds, prompted him to ask friends and family to raise the head end of their beds on bricks, blocks or books to a five-degree angle to see what would happen. Within four weeks two volunteers reported back significant improvements in varicose veins.
This again meant that the literature needed serious review, because patients who were being advised to put their feet up higher and now placed their feet lower, were responding to what must be the simplest non-invasive method of study available. A raised bed at the head end position seemed to improve wellbeing.
Andrew’s mother was the first to report being able to move her ankle and wiggle her toes for the first time in over ten years after only four weeks of IBT. Later her sensation in the limb became normal.
Following more compelling reports from patients who trialled IBT, there became an inevitable positive change for people with neurological conditions, which were believed to be irreversible. People with Multiple Sclerosis (MS) were observing substantial improvements.
The argument against Andrew's results were that MS could relapse and remit, despite there being reports from people with primary progressive MS. This argument was thwarted when he extended his theory and research to patients with complete spinal cord injury, who also reported significant improvements in bowel and bladder control, sensation and movement.
Successes prompted Andrew to research cerebrospinal fluid circulation and the onset and lack of progress in many neurological problems, including Parkinson’s Disease, Cerebral Palsy, Spinal Cord Injury, Sciatica, Fibromyalgia, Migraine Headaches, Strokes, Comatose patient care, restless leg syndrome , broken bone growth and many more conditions, all of which appeared to benefit from IBT.
An independent report from the Multiple Sclerosis Resource Centre (MSRC), was conducted by John and Jean Simkins, titled: Raised Bed Survey, also published here. Documented how ten people with MS and two people with spinal cord injury positively progressed using IBT.
Andrew’s battle to alert the medical profession and science communities to his findings, inviting them to challenge his discovery by testing the results, instead of dismissing them out of hand as being anecdotal evidence, has been relentless.
A lesser man would have given up long ago. But Andrew is driven on to help many more people, with or without ‘academic recognition’ to establish the truth.
History of Beds
Andrew’s research into the history of beds and how we used to sleep took him to rediscover how the Pharaohs and their followers slept inclined in Ancient Egypt. Those ancient beds are in museums around the world and no one is asking why our ancestors slept inclined. Andrew also found images of inclined beds in an ancient Constantinople hospital and C sections carried out successfully using an inclined bed, by natives in Africa. Images of babies swaddled in cots and cribs were also found, indicating a long since forgotten wisdom.
Astronauts and Space Travel
Andrew asked what happens to our physiology when we enter orbit in microgravity. He Found a great deal of evidence from various Space Research Studies, which used prolonged flat bed rest and head down bed rest as a model to replicate the rapid ageing, which according to Joan Vernikos, former head of NASA Life Sciences, is 10 times faster than here on Earth.
These prolonged bed rest trials revealed that the same degenerative processes experienced by astronauts were present in healthy young people who were confined to bed and studied for many months. Astronauts experience heart atrophy, fluid shift, bone decalcification, osteoporosis, muscular atrophy, skin thinning, psoriasis and other skin conditions, sight degeneration, immune deficiency, kidney stones, liver stones, bladder stones, urine infections, circulatory disorders, vestibular disorders, chronic inflammation, sleep disorders, accumulation of carbon dioxide, lymphatic disorders and more. On return to Earth, they are unable to walk or even stand upright.
Studies, Work and Documentation
Andrew had documented and shared his results in mainstream media, including the Daily Mail, Woman’s Realm, BBC World Service, Carlton TV News and many more publications.
At the London International Inventions Fair in 1997 his inclined bed invention won a gold award after he demonstrated a circulation model in tubes that showed clearly how circulation in humans and animals benefit from the direction of gravity.
He invited visitors, including many scientists, doctors, inventors and the public to lay on his inclined bed to determine the differences between sleeping flat against sleeping at an incline. Most could determine the differences immediately.
Andrew has documented many of his early fascinating news cuttings, letters (including support and rejections by doctors), and articles from both science journals and charity sites on his website. These are supported with a compelling bank of evidence, including astonishing testimonials and case histories.
Andrew has continuously campaigned to have clinical studies conducted to test Inclined Bed Therapy results to establish IBT in hospitals to improve patient recovery and survival rates. The simple modification to a bed can follow the patient home and continue to aid recovery from surgery and prevent many complications, improving patients’ lives and saving overburdened health services a great deal of resources, time and money.
Diabetes Trial Success.
One such study has been conducted into Diabetes, which showed that IBT lowers blood sugar levels and helps with many other diabetes associated symptoms.
INCLINED BED THERAPY: NEW HOPE FOR DIABETES By Erica Billen Pohnpei Island Central School & Tetaake Yee Ting College of Micronesia-FSM Pohnpei, Micronesia
To put an end to Sudden Infant Death Syndrome (SIDS)
To put an end to SIDS by encouraging parents to place babies on an inclined mattress of no more than 5 degrees. Andrew met with Dr Chantler, senior medical research advisor to the Foundation for Sudden Infant Death Syndrome, now named the Lullaby Foundation and convinced her that the science was correct. After hearing Andrew, and seeing his experiments at her home in London, he proved he had solved SIDS! And yet to – date, no further research has been forthcoming.
To put an end to Sudden Unexplained Adult Deaths (SADS) Which appears to mirror (SIDS)
Healthy adults are going to bed, never to wake up? It should tell us all something about the dangers of bed rest?
To save patients from losing limbs and in some cases their lives to gangrene, leg ulcers, pressure sores and many other infections, including pneumonia, scarlatina, thrombosis,
To Introduce IBT as normal practice in aiding and speeding up recovery in all hospitals.
To spread the word about IBT in developing countries affected by AIDS and many other debilitating conditions.
“Reward a child for thinking and not just for remembering and regurgitate someone else’s beliefs. Then we will see foundations crumble and brick walls fall!” Andrew K Fletcher
When asked why do you continue? Andrew will always reply: “Because it is the right thing to do!”.
McKean, DG. GCSE Biology. Hodder Murray, 1989
BBC World Service
Carlton TV News
London International Inventions Fair, 1997
‘those text books…showed direction of blood flow ….’ P3
Cite actual studies?
Thwarted argument re R&R MS
Multiple Sclerosis Resource Centre MSRC John and Jean Simkins: Raised Bed Survey
Nature Journal: Scientific Reports, Corrigendum
Egypt / Africa ref
We do hope that you enjoy your stay here and that you find this information helpful.
Do check out the testimonials in the top menu, along with watching the video interviews with Andrew explaining, in detail, how his exiting sleep discovery began in 1994.
If you have something to share or a question to ask, Andrew will be happy to help. If you have been using IBT Please write your testimonial and share your experience to help others realise their own health benefits.
Consider helping in this important research by reporting back to us via email or in the forum. andrewkfletcher AT gmail.com
Help to spread this IBT message by sharing your experience with friends and family.
AndrewAdd a comment